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Danazol appears to exert its principal pharmacological activity in the endocrine system as an inhibitor of gonadotrophin synthesis and/or release by the pituitaryThe mechanisms by which danazol exerts its therapeutic effect are incompletely understood and there are considered to be four possible mechanisms of the action as follows:
In view of its pharmacologyknown interactions and side effectsparticular care should be observed when using danazol in patients with hepatic or renal diseasehypertension or other cardiovascular disease and in any state which may be exacerbated by fluid retention as well as in diabetes mellituspolycythaemiaepilepsylipoprotein disorderin those with a history of thrombosisand in those who have shown marked or persistent androgenic reaction to previous gonadal order periactine from indian. steroid therapyAdjustment in concomitant therapy may be called for particularly in patients with hypertensiondiabetes mellitus or epilepsy when introducing or discontinuing danazol as well as during Danazol treatment.
Sudden numbness or weaknessespecially on one side of the bodyconfusionproblems with speech or balancesevere headacheringing in your earsdizzinessnauseavomitingvision problemspain behind your eyeschest painsudden coughwheezingrapid breathingcoughing up bloodpainswellingwarmthor redness in one or both legsswelling or rapid weight gainespecially in your face and midsectionbloodyblackor tarry stoolscough up blood or vomit that looks like coffee groundsor upper stomach painitchingloss of appetitedark urineclay-colored stoolsjaundiceyellowing of the skin or eyes
In infrequent patientssymptoms of pain and tenderness may be severe enough to warrant treatment by suppression of ovarian functionDanazol is usually effective in decreasing nodularitypainand tendernessIt should be stressed to the patient that this treatment is not innocuous in that it involves considerable alterations of hormone levels and that recurrence of symptoms is very common after cessation of therapy.
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vero-danazol can increase the calcemic response to synthetic vitamin d analogs in primary hypoparathyroidism.0